CONCLUSION Free radical produced by self-oxidation of bendopa can promote development of Parkinson disease. APS can relieve the toxicity of bendopa on neurons and the effect has time dependency. 结论:左旋多巴自身氧化产生大量的自由基,促进帕金森病发展,黄芪多糖可降低左旋多巴对神经元的毒性作用,作用有时间依赖性。
And the dosage of bendopa preparation was compared before and after operation. 并对术前术后左旋多巴制剂用量进行了比较。