anergy

1. Howeer, some T cells are not actiated, in fact they are inactivated by a process called anergy or tolerance.
   然而，有些T细胞处于未激发状态，事实上他们被称为无反应性，耐受性的过程灭活了。
2. Inhibition of this pathway results in development of antigen-specific unresponsiveness and clonal anergy.
   阻断该通路可以导致抗原特异性的免疫耐受和克隆不应答状态。
3. Introduces the concepts of available anergy and reduced exergy by analysing the energy utilization process.
   通过对其能量利用过程的分析，引入可用和折合的概念。
4. Recently, MSC have been shown to exert a profound immunosuppressive effect on polyclonal as well as antigen-specific T cell responses by inducing a state of division arrest anergy.
   近来认为，MSCs通过诱导分裂停滞无反应性来对多克隆和特异抗原T细胞反应有明显的免疫抑制作用。
5. Given that severe trauma effects as many as20 percent of the20,000 or so human genes, finding the genes and proteins involved in T cell anergy and apoptosis amid this storm has been a challenge.
   如果大面积损伤影响多达人类基因（大概20000个）中20%的基因的话，找到其中涉及T细胞无能和凋亡的基因和蛋白是一个挑战。
6. Superantigen staphylococcal enterotoxin A induces anergy of T lymphocytes in vivo
   超抗原葡萄球菌肠毒素A对体内T细胞无能的诱导规律
7. The simulation showed that LEACH-SERP Protocol could effectively save anergy, balance network load and extend the network's lifetime.
   仿真结果表明，LEACH-SERP协议有效节约能量，平衡网络负载，延长网络生存时间。
8. While the TCR engagement with specific antigen, the administration of CTLA-4Ig can induce T cell anergy in vitro or tolerance in vivo.
   在TCR识别外来抗原的同时给予可溶性CTLA-4，在体外可诱导T细胞无应答，在体内则可诱导特异性免疫耐受。
9. Objectives To study the induction and tolerance properties of antigen specific T cell anergy.
   目的探讨抗原特异性无能T淋巴细胞的诱导及其耐受特性。
10. Aim To explore the molecular mechanism of T cell anergy induced by superantigen SEA.
    目的探讨超抗原诱导T细胞无能的分子机制。
11. The result of IL-2 reverse test showed that clone anergy was probably involved in the 'formation of tolerance in the tolerant B6 mice.
    IL2逆转实验结果表明克隆无能参与了移植耐受的形成。
12. Objective: Study the correlation between T cell anergy and apoptosis.
    目的：探讨无能T细胞与凋亡的关系。
13. Conclusion The decrease of IL 2 level and the increase of IL 10 level probably contribute to the induction of T cell anergy.
    结论超抗原SEA对T细胞无能的诱导，可能与降低IL2的水平和升高IL10的水平有关。
14. Multiple mechanisms, including clone anergy and suppressor cells were involved in the tolerence.
    多种耐受机制，包括克隆不应答、抑制细胞，都参与了耐受的形成。
15. CD47 induces costimulatory signals on activation of T cell, T cell apoptosis and T cell anergy and enhances the efficiency of TCR signaling;
    CD47是T细胞活化的共刺激因子，活化T细胞凋亡过程，诱导T细胞无反应性，加强TCR信号传导的效率；
16. Molecular mechanism of Tcell anergy induced by superantigen SEA
    超抗原SEA诱导T细胞无能的作用机制探讨
17. The results from the present study may have important implications for the study of the mechanism ( s) underlying T cell anergy induction in vivo and for applications of immune tolerance based therapy.
    这对体内T细胞免疫耐受产生的机制研究和临床应用都有重要意义。
18. Study on the Relationship Between Costimulatory Molecules CD28/ CTLA-4 and T Lymphocytes Anergy Induced by Superantigen SEA
    CTLA-4与超抗原对SEA诱导T细胞无能的关联研究
19. T cell anergy has been successfully induced under different conditions in cloned CD4+ T cells, but induction of T cell anergy in vivo has been difficult and controversial.
    在体外克隆T细胞中，T细胞无能可在多种条件下诱导产生，但T细胞在体内条件下的无能诱导仍有很多疑问和争议。
20. Anergy and Characteristics of Human T Cells Induced by Combination with B7-1 mAb and CsA
    B7-1单抗和CsA联用诱导人T细胞无能及其特性探讨
21. To study the induced condition and characteristics of T cell anergy in vitro.
    目的：在体外探讨T细胞无能诱导的条件及其生物学特性。
22. T cell anergy during Helicobacter pylori infection stems from the apoptosis mediated by Fas/ FasL interaction
    幽门螺杆菌致T细胞凋亡：Fas/FasL介导的T细胞无反应性
23. Clone anergy, suppressive cells and chimerism were probably involved in the formation of tolerance.
    克隆不应答、抑制细胞和嵌合体的形成等机制都参与了耐受形成。
24. Non-polarity, low surface anergy and weak surface layer, etc. may be the major causes of the lower painting adhesion of polyolefine plastics, such as polyethylene, polypropylene, polystyrene and teflon.
    分析了聚烯烃塑料，诸如聚乙烯、聚丙烯、聚苯乙烯和聚四氟乙烯涂装附着性低的原因&无极性、低表面自由能和弱界面层存在等。
25. Without sufficient co-stimulation, T cells are rendered anergy, tolerance or apoptosis.
    如果没有足够的共刺激信号刺激，T细胞表现为失能、耐受和凋亡。
26. However, inhibitory costimulatory factors could be involved in the pathogenesis in two ways by inducing T cell anergy: inducing inhibitory cofactor expression in effector immune cells or expression of ligands of inhibitory cofactor on tumor cells.
    负性共刺激分子通过两种方法来诱导T细胞无力：一是诱导抑制性因子表达在效应性免疫细胞上，二是诱导抑制性因子受体表达在肿瘤细胞上。
27. Immature DCs have lower levels of co-stimulatory molecules and usually induce antigen-specific T cell anergy.
    ImDCs共刺激分子表达更低，通常诱导抗原特异性的T细胞失能。

noun

1. inactivity and lack of energy

2. reduction or lack of an immune response to a specific antigen