atherogenesis

1. Objective To observe the progress of atherogenesis in vein-grafts and explore relationship between variation of proteoglycans ( PGs) and progress of atherogenesis.
   目的观察移植静脉粥样化过程，探讨蛋白聚糖（PG）对粥样化改变的影响。
2. Contrast study of vascular intimal and adventitia injury in atherogenesis and signal pathway mechanism
   损伤血管内、外膜致动脉粥样硬化形成及信号转导机制的对比研究
3. Although the participation of inflammation in atherogenesis is widely recognized, the identification of the different components has not been clarified.
   背景-尽管动脉粥样硬化中有炎症因素参与是广为认同的，但对于不同（炎性）成分的确认还不甚明了。
4. Oxidative stress is implicated in atherogenesis, yet most clinical trials with antioxidants, particularly vitamin E, have failed to protect against atherosclerotic diseases.
   氧化应激与动脉粥样硬化有关，然而大量临床研究表明抗氧化剂，特别是维生素E并不能预防动脉粥样硬化性疾病。
5. Role and Mechanisms of Sympathetic/ Parasympathetic Neurotransmitters in Atherogenesis
   交感/副交感神经递质在动脉粥样硬化病变发生中的研究
6. The effect of Ras blocking agent on early atherogenesis in rats with diabetes mellitus
   RAS阻断剂对糖尿病大鼠早期动脉粥样硬化形成影响
7. Does Infection Play a Role in Atherogenesis and Plaque Rupture?
   动脉硬化与斑块破裂：感染是否在起作用？
8. Although some data from experimental models support a role of CRP in atherogenesis, its direct involvement remains in question.
   尽管由实验模型得到的数据支持CRP在动脉粥样硬化形成中的作用，但它的直接关系仍在讨论中。
9. Atherogenesis mechanism is constantly studied by cardiovascular scholar.
   动脉粥样硬化（Atherosclerosis，AS）是严重影响人类健康的疾病之一，其发生机制一直是心血管领域的研究热点。
10. Objective: To investigate the effect of Astragalus Polysaccharide ( APS) on the function of endothelial cells in atherogenesis rabbits.
    目的：探讨黄芪多糖对新西兰兔动脉粥样硬化内皮细胞功能的影响。
11. Objective To investigate the effect of heat shock protein 60 ( HSP60), an important inflammatory factor in atherogenesis, on the maturation of murine dendritic cells ( mDCs) in vitro.
    目的探讨动脉粥样硬化中重要炎性物质&热休克蛋白60（HSP60）体外对小鼠树突状细胞（mDC）功能的影响。
12. The results showed that fish oil could obviously decrease triglyceride in serum and have no effect on atherogenesis by angiography.
    结果表明，鱼油制剂能明显降低血清中甘油三酯水平，血管造影提示对动脉粥样硬化形成无影响。
13. NAD ( P) H oxidase is the principal source of reactive oxygen species in neutrophilic leukocyte and vascular system, and plays an important role in the ischemic lesion and atherogenesis ( As).
    NAD（P）H氧化酶是中性粒细胞和血管系统中的活性氧主要来源，在缺血性损害和动脉粥样硬化形成中发挥了重要作用。
14. The dynamic change of extracellular matrix in human coronary atherogenesis
    人冠状动脉几种细胞外基质的动态变化与动脉粥样硬化形成的关系
15. Effect of lovastatin on atherogenesis and expression of related oncogenes in experimental rabbits
    洛伐他汀对实验性家兔动脉粥样硬化形成及其相关癌基因表达作用的研究
16. Conclusion: Hcy may contribute to atherogenesis by induction of IL-8 secretion in THP-1 macrophage.
    结论：Hcy能促进人THP-1巨噬细胞IL-8的表达和分泌，可能为其诱导动脉粥样硬化的重要机制之一。
17. CONCLUSION: COX and its products play an important role for plaque vulnerability at the different stages of atherogenesis.
    结论：环氧合酶及其产物在动脉粥样硬化发展的不同阶段，对于斑块稳定性起着重要作用。
18. The Effect and Mechanism of Pulsatile Low Shear Stress on Atherogenesis
    脉动低切应力对动脉粥样硬化形成的影响及其机制
19. Relationship between distribution and change of heparan sulfate proteoglycan and atherogenesis in human coronary arteries
    人冠状动脉硫酸乙酰肝素蛋白聚糖的分布和变化与动脉粥样硬化形成的关系
20. The Role of the Blood-borne Monocyte in Atherogenesis in Rabbits
    血源性单核巨噬细胞在兔动脉粥样硬化发生中的作用
21. Objective: To study the modificatory effect of macrophages on low density lipoprotein ( LDL) and the inhibitive effect of prostaglandin E2 ( PGE2) on atherogenesis.
    目的：研究巨噬细胞对低密度脂蛋白（LDL）的修饰和前列腺素E2（PGE2）对动脉粥样硬化形成的抑制作用。
22. Objective To study the effect of fluid shear stress on the expression of monocyte chemotactic protein 1 ( MCP 1) by vascular endothelial cells and its role in the early stages of atherogenesis ( AS).
    目的研究流体切应力对人血管内皮细胞的单核细胞趋化蛋白1（MCP1）表达的影响，探讨血流动力在动脉粥样硬化（AS）发生早期中的作用。
23. Objective To study the mechanism of monocyte recruitment in atherogenesis and to clarify the effect of monocyte chemotactic protein 1 ( MCP1) in this process.
    目的了解动脉粥样硬化发病过程中单核细胞粘附并进入动脉内膜的机制，分析单核细胞趋化蛋白1（MCP1）基因表达对单核细胞粘附动脉内膜的作用。
24. Objective To isolate and clone the differentially expressed genes in vascular endothelial cell induced by high density lipoprotein ( HDL) so as to study the anti atherogenesis molecular mechanism of HDL.
    目的研究高密度脂蛋白（HDL）抗动脉粥样硬化（AS）的分子机理，克隆并分析血管内皮细胞在HDL作用下差异表达的已知和未知基因。
25. The Hp CagA+ strain infection may promote and aggravate carotid artery atherogenesis through enhancement inflammation response and immunity in plaque.
    HpCagA+菌株感染者，可能通过增强粥样硬化斑块局部的炎症反应及免疫反应促进和加重颈动脉粥样硬化形成。
26. First, the mechanisms of atherogenesis have not been fully elucidated.
    一方面，动脉粥样硬化的发生机制至今未被完全阐明。
27. There are many research areas in studies of atherosclerosis, among which atherogenesis and characterization of vulnerable plaques are of great importance.
    在动脉粥样硬化的研究中，有两项重要内容：发生机制的研究与易损斑块的识别。
28. Endothelial dysfunction, infiltrating of leukocytes, oxidative stress, cell apoptosis, lipid accumulation, angiogenesis, collagen content and many other factors contribute to atherogenesis.
    血脂异常、内皮功能障碍、炎症细胞浸润、氧化应激、细胞凋亡、脂质沉积、血管生成、胶原代谢等因素均在动脉粥样硬化中发挥了一定的作用。
29. Vascular smooth muscle cells play a very important role in atherogenesis, proliferation, migration and producing extracellular matrix are the essential characteristics of it.
    平滑肌细胞在动脉粥样硬化形成过程中起到很重要的作用，平滑肌细胞的增殖、迁移和分泌细胞外基质是动脉粥样硬化发生发展的基本特征之一。

noun

1. the formation of atheromas on the walls of the arteries as in atherosclerosis