Objective: To study the effect of Shenkang injection on the proliferation of human mesangial cell induced by LPS. 目的:探讨中药肾康注射液对脂多糖诱导肾小球系膜细胞增殖的影响。
The Effect of Atorvastatin on TNF-α and IL-1 β in Peripheral Blood Monocytes Stimulated by LPS 阿托伐他汀对脂多糖刺激下外周血单核细胞分泌TNF-α、IL-1β的影响
Methods the hepatic model of LPS-induced acute inflammatory injury was established by administrating LPS and bcg. 方法采用卡介苗(bcg)配合lps建立小鼠急性炎性肝损伤模型;
AM-Pharma is developing a unique therapy against sepsis, based on detoxification of the bacterial endotoxin LPS. Pharma公司正基于细菌性内毒素LPS的去毒作用,研究一种专门治疗败血症的方法。
However, the relationship between PAR-2 signaling pathway and LPS-activated pro-inflammation response remains unclear. 然而,PAR-2讯息路径和LPS活化的促发炎反应之间的关系仍不清楚。
Role of endogenous and exogenous hydrogen sulfide in acute lung injury induced by LPS in rats 内、外源性硫化氢在脂多糖所致大鼠急性肺损伤中的作用
In a word, S-adenosylmethionine may be beneficial to LPS-induced liver in-jury in clinical treatment. S-腺苷蛋氨酸的这些作用可能有益于LPS诱导的肝损伤的临床治疗。
Protective Effect of AF-2 on LPS-induced Acute Lung Injury of Mouse 抗炎多肽AF-2对LPS诱导的小鼠急性肺损伤的保护
The Effects of NF-κ B on Parkinson Disease Rat Model Induced by LPS NF-κB在LPS诱导的帕金森病模型大鼠中的作用
The LPS activating effect of HUVEC membrane lipid showed a typical property of enzymatic dynamics. LPS激活膜磷脂代谢的反应特性表现出典型的酶促动力学特性。
The Protective Effect of PDTC on Acute Lung Injury Induced by LPS in Rats PDTC对LPS诱导大鼠急性肺损伤的保护作用
We hypothesize that reactive oxygen species ( ROS) mediate lipopolysaccharide ( LPS)-evoked hepatic lipid accumulation. 我们假定活性氧簇分子参与了脂多糖诱导的肝细胞脂肪堆积。
Effect of aminoguanidine on pulmonary surfactant and alveolar macrophages in the rats induced by LPS 氨基胍对脂多糖诱导的大鼠肺表面活性物质和肺泡巨噬细胞的影响
They first showed that stimulation with lipopolysaccharide ( LPS) and concanavalin A increased tonsillar and adenoidal proliferation in cells from OSA children compared with controls. 他们首先显示出在脂多糖和刀豆蛋白A的刺激下,OSA儿童较糖皮质激素控制组的扁桃体和腺样体的细胞增殖增多。
A small dose of lipopolysaccharide ( LPS) was administered intraperitoneally once to aggravate endotoxemia of animal with cirrhosis. 腹腔内一次性注射小剂量内毒素以加重肝硬化动物的内毒素血症。
ConclusionThe soluble factors released by Kupffer cells, which were activated by LPS, could cause direct hepatocytes injury. 结论内毒素诱导库普弗细胞释放的可溶性因子作用一定时间后可直接对肝细胞造成损伤。
Treatment of cells with the TLR-4 agonist, LPS, did not reduce trophoblast cell viability. 而以TLR-4激动剂LPS处理的滋养层细胞并未有细胞活性下降。
Preliminary analysis on relationship between the TNF secretion in rat macrophages and the LPS dose 大鼠巨噬细胞肿瘤坏死因子的分泌与内毒素剂量关系的初步分析
Protective Effect of Insulin on LPS-induced Injury of Normal Human Liver Cells 胰岛素对LPS诱导的人肝细胞损伤的保护作用
At the same time, the performance of LPS neutralization in kidney of macaques is up-regulated. 在此同时,肾脏实质细胞内中和内毒素的作用也增强。
Effects of Melatonin on the Antioxidant and Immune Function of Mice under the Stimulation of LPS 褪黑素对脂多糖刺激小鼠抗氧化与免疫功能的影响
Conclusion LPS could involve bone tissue in inflammation and inhibit the differentiation of osteoblast. 结论LPS刺激骨组织发生炎症反应、抑制成骨细胞分化。
Effects of tilmicosin on cytokine response and survival rate of inflammatory mice by LPS 替米考星对内毒素炎症小鼠存活率和细胞因子的影响
Suppressive effects of hydroxytyrosol on oxidative stress of THP-1 cells stimulated by LPS 羟基酪醇对LPS刺激的THP-1细胞氧化应激的抑制作用
The Research of Lipopolysaccharide on NF-κ B Expression and Inflammatory Cytokines Production in Human Periodontal Ligament Cells LPS对人牙周韧带细胞核转录因子-κB表达及炎性细胞因子分泌的研究
Objective: To observe the effects of the root of Chinese Pulsatilla ( RCP) on the secretion of TNF, IL ˉ 1and IL ˉ 6from Kupffer cells ( KC) stimulated by Lipopolysaccharide ( LPS). 目的:观察白头翁对抑制脂多糖(LPS)刺激肝枯否氏细胞(KC)分泌肿瘤坏死因子(TNF)、白细胞介素1(IL1)和白细胞介素6(IL6)的影响。
There are evidences that several Gram-negative bacterial strains use their core LPS to interact with C-type lectin receptor. 由于近来发现的C型凝集素受体与某些革兰氏阴性菌的核心脂多糖(核心LPS)有作用。
Experimental Study of the Effect of NF-κ B on Endothelial Cells Apoptosis Induced by LPS NF-κB对内毒素诱导的血管内皮细胞凋亡的影响
Objective To investigate the protective effect of ACTH ( 1-24) on ECV304 damaged by LPS. 目的研究ACTH(1-24)对脂多糖损伤的脐静脉内皮细胞(ECV304)的保护作用。