This could be a simple staph infection causing neurodegeneration. 或许这只是葡萄球菌感染造成的神经退变。
The present review focused on the application of biodegradable delivery systems of growth factors in treating neurodegeneration diseases. 就生物可降解生长因子缓释制剂在神经系统退变性疾病中的应用做一综述。
For example, autophagic dysfunction is associated with cancer, neurodegeneration, microbial infection and ageing. 例如,自噬功能障碍与癌症,神经退行性病变,微生物感染和老化相关。
AIM To explore whether there is relationship between iron homeostasis disorder and neurodegeneration induced by aluminum ( Al) overload. 目的探讨铝致神经元退行性变是否与脑铁代谢失衡有关。
The Relationship of Calcium Homeostasis and Oxidative Stress in Neurodegeneration 神经变性过程中钙稳态变化与氧化应激初步研究
The role of autophagy-lysosome pathway in neurodegeneration associated with Parkinson's disease. 帕金森病相关神经变性中自噬溶酶体途径的作用。
Protective Effects and Mechanisms of Meloxicam on Chronic Aluminum Overload-Induced Neurodegeneration of Rats 美洛昔康对慢性铝过负荷致大鼠神经元退变的保护作用及机制
Autophagy principally serves an adaptive role to protect organisms against diverse pathologies, including infections, cancer, neurodegeneration, aging, and heart disease. 自噬主要起保护生物体抵抗多种多样病理的自适应作用,包括感染、肿瘤、神经变性、老化和心脏病。
However, there are currently no treatments available that slow the neurodegeneration in people with Huntington's disease. 然而,现在临床上还没有可用的减缓亨延顿舞蹈综合征神经退行性变的方法。
Recently clinical and preclinical study indicated that neurodegeneration happened earlier than capillary vessel changes in the retina of diabetic patients and animal model. 但是近年来的临床和基础研究发现糖尿病患者和动物模型在视网膜特征性微血管发生改变以前已经出现了神经元的改变。
For example, in recent years scientists have become increasingly aware of the importance of mitochondrial function in treating diseases such as cancer, diabetes, and neurodegeneration. 例如,近年来,在治疗像癌症、糖尿病以及神经退化性疾病时,科学家越来越注意到线粒体功能的重要性。
This article reviews the recent perspectives on retinal neurodegeneration in the early stage of diabetes. 现将糖尿病视网膜病变早期视网膜神经退行性病变的国内外研究进展综述如下。
Dysfunction of the UPS has been already strongly implicated in the pathogenesis of this disease and, more recently, growing interest has been shown in identifying the role of ALP in neurodegeneration. 这些系统降解错折叠和聚集蛋白的功能改变在比如帕金森病的许多神经变性疾病中起关键作用已逐渐被认识。
The finding sheds light on the molecular mechanism by which cells compute their fate, and may point to therapeutic targets that protect against or delay the onset of neurodegeneration. 研究揭示了细胞自我控制的凋亡过程的相关分子机制,并有可能发现防止或延迟神经退行性病变的治疗靶点。
Past adult lead exposure is linked to neurodegeneration measured by brain MRI 成年铅暴露史与脑部MRI检测的神经退行性变有关
This paper attempts to review the strategies to protect the neurodegeneration that occurs in both Parkinson's and Huntington's diseases. 本文就近年有关成功减轻帕金森病和亨廷顿病中的神经退行性病的神经保护策略进行综述如下。
Conclusions Deposits of β-AP in brains might be related to neurodegeneration, intellectual decline and AD. 结论β-AP在脑中的沉积可能与神经元的变性以致智能衰退、AD的发生有关。
Conclusion: Excitotoxic may be an important mechanism for trauma-induced neurodegeneration in immature brain. 结论:内源性兴奋毒对未成熟脑创伤性神经元变性起十分重要的作用。
Role of Astrocyte in the Aetiology of Neurodegeneration Diseases Involving Inflammation 星形胶质细胞在炎症相关的神经变性疾病发病机制中的作用研究
Metabolic compromise, excitotoxicity, and oxidative stress are three main mechanisms of neuronal cell death, they may act separately or cooperatively to cause neurodegeneration. 神经细胞死亡的3种机制为代谢障碍、兴奋性毒性及氧化应激,它们可以独立或相互作用而导致神经退行性变的发生。
Okadaic acid induces tau protein phosphorylation and neurodegeneration in rat brain 冈田酸诱导大鼠脑tau蛋白磷酸化和神经细胞退化
Study of the relationship between aluminum overload-induced neurodegeneration and systems of oxidative stress and anti-oxidative stress in mice 铝过负荷致小鼠神经退行性变与氧化应激和抗氧化应激系统关系的研究
A vast of studies has indicated quercetin can physiologically protect neurons and alleviated the progression of neurodegeneration diseases through altering related gene expression in astrocytes. 很多研究已经表明槲皮素可以通过改变反应性星形胶质细胞基因的表达来保护神经元和减少神经变性疾病的发生发展。
Iron, a hemoglobin degradation product, plays a key role in neurodegeneration in many disease states, and an increase in brain iron can result in lipid peroxidation and free radical formation. 铁,作为血红蛋白的降解产物,在许多疾病的神经损害中起着关键作用,脑内铁含量的增加可导致脂质过氧化和自由基产生。
It has been reported that ethanol exposure leads to impairments of the cerebra and cerebellum in different areas, and triggers widespread apoptotic neurodegeneration in infant rat and mouse brain. 在神经退行性疾病研究中,动物实验表明,酒精可导致大脑与小脑不同区域的损伤,并广泛的触发神经细胞退行性凋亡。
So it is possible for transplanted neurons to integrate into neuronal networks composed by host neurons, repairing the lost function caused by neurodegeneration or CNS injury. 这就使得一种设想成为可能:植入的神经元加入到宿主神经元形成的神经网络中,修复由于神经变性和损伤导致的神经功能的缺陷。