ostarthritis
英
美
n. 骨关节炎
双语例句
- The interleukin-18, newly found factor-induced inflammation, was essential pre-inflammatory factor in the pathogenesy of ostarthritis, could induce the production of prostaglandin E2. The effect of interleukin-18 and prostaglandin E2 in osteoarthritis pathogenesis were revealed in the trials of immune, pathology and animals.
新发现致炎因子白细胞介素18是骨关节炎发病机制中重要的一种炎症前因子，能诱导炎症介质前列腺素E2的产生。 - Clinical research and application of autologous chondrocyte implantation for knee ostarthritis
自体软骨细胞移植治疗膝骨关节炎的临床研究与应用 - CONCLUSION: Interleukin-18 and prostaglandin E2 take part in pathogenesy of ostarthritis.
从免疫、病理、动物试验进行研究，揭示白细胞介素18，前列腺素E2在骨关节炎发病机制中的作用。 - Of the 48 articles, 19 articles were on chondrocytes cultured in vitro, 16 articles were on incidence of the knee ostarthritis and Chinese medicine preventing the articular cartilage cataplasia, and 13 articles were prospects and historical background of articular cartilage defects repaired by transplantation of chondrocytes.
48篇文献中有关软骨细胞体外培养的文章19篇，膝骨关节炎的发病率及中医药防治关节软骨退变的文章16篇，软骨细胞修复关节软骨缺损的历史及前景展望的文章13篇。 - The increase of interleukin-18 maybe causes the increase of the content of prostaglandin E2, so as to play important role in the nosogenesis of ostarthritis.
结论：白细胞介素18与前列腺素E2参与了骨关节炎的发病机制，白细胞介素18的增高可能引起前列腺素E2含量的增高，从而在骨关节炎的发病机制中发挥重要作用。 - Interleukin-18 and prostaglandin E2 in ostarthritis
白细胞介素18和前列腺素E2与骨关节炎