pannus

美 [ˈpɑnuz]

n.  血管翳,角膜翳,关节翳

医学



双语例句

  1. Pannus is a form of superficial keratitis, or inflammation of the cornea, found most commonly in German Shepherd Dogs, Greyhounds, and Siberian Huskies.
    角膜血管翳是一种浅表性角膜炎,常见于德国牧羊犬、意大利灵缇犬和哈士奇。
  2. The warming and activating effect of moxibustion therapy may involve the inhibition of the formation of ICAM-1 and pannus.
    艾灸能抑制血清细胞粘附分子ICAM-1的产生,阻抑血管翳生成,可能是艾灸温经通络治疗实验性关节炎的作用机制之一。
  3. Analysis of the location of intraoperative corneal pannus hemorrhage during LASIK in 48 cases
    LASIK中角膜血管翳的出血位置分析
  4. Synovitis, the hyperplasia of synovium, pannus formation and the destruction of cartilage and bone are the main pathohistological properties of RA.
    滑膜炎症,滑膜组织增生和血管翳形成,关节软骨和骨组织结构的破坏,是RA的主要病理组织学改变。
  5. Results No pathological change was noticed in the control group. Among the 96 cases of SpA, pathological changes were found in 76 cases, including bone marrow inflammation, pannus formation, subchondral bony plate destruction, cartilage degeneration/ erosion, synovitis, and enthesitis.
    结果76例SpA的SIJ存在炎症表现,包括骨髓炎、血管翳形成和炎性细胞浸润,软骨下骨板破坏,软骨变性、破坏,滑膜炎,附着点炎等。
  6. The prominent pathologies of RA are observed: synovial infiltration of multiple inflammatory cells mainly composing of T lymphocyte, synovial lining cells proliferation, angiogenesis and pannus formation.
    RA的滑膜病理学表现以T淋巴细胞为主的大量炎细胞浸润、滑膜衬里层细胞增殖、血管新生及血管翳形成。
  7. Histopathologic features of RA encompass infiltration by macrophages and T cells synovial lining hyperplasia, neoangiogenesis, pannus formation and destruction of cartilage and bone.
    其主要病理特点是滑膜细胞增生、衬里层增厚、多种炎性细胞浸润、血管翳形成,以及软骨和骨组织的破坏。
  8. Power Doppler sonography in assessment of synovial pannus of rheumatoid knee after~ ( 32) P radiation synovectomy
    类风湿性膝关节炎放射性滑膜切除治疗后血管翳变化的能量多普勒研究
  9. Which is different from RA. Conclusion The expression of VEGF is relative to the synovial pannus formation in RA.
    结论:VEGF参与了RA疾病滑膜炎的形成和发展过程;其VEGF表达的高低与关节炎症状的轻重有关。
  10. In the CT ⅱ and ⅲ degree sacroiliitis, marked degeneration and fibrosis of cartilage, inflammatory cell infiltration, pannus formation and subchondral bony plate destruction were increasingly prominent.
    CTⅡ~Ⅲ级SIJ炎病理变化主要为软骨严重变性、破坏,血管翳形成,大量炎症细胞浸润,软骨下骨板破坏;
  11. The diagnosis was ichthyosis in histopathological examination, congenital nerve deaf-mutism in otolaryngology and corneal pannus in ophthalmology.
    躯干部皮肤明显干燥,皮肤角化过度,呈皮革样变化。病理活检为鱼鳞病,耳科检查为先天感音神经性耳聋,眼科检查为角膜血管翳。
  12. BACKGROUND: Pathological change of synovium in rheumatoid arthritis RA has the characteristic of tumor-like growth, it appears thickening of the synovium tissue and the formation of pannus, which generate periarticular erosion and destruction.
    背景:类风湿关节炎的滑膜病变具有类肿瘤样生长的特点,表现为滑膜组织的肥厚增生,血管翳形成,对关节周围的组织产生侵蚀和破坏。
  13. The main changes including synovial hyperplasia, pannus formation, joint effusion, articular cartilage destruction, bone erosions, meniscus and ligament lesions, popliteal cysts and subcutaneous nodules formation were depicted clearly by MR imaging.
    主要影像学表现包括滑膜增生与血管翳形成、关节囊积液、关节软骨与骨质受侵破坏、半月板与韧带异常、腘窝囊肿及皮下结节形成等。
  14. Results: Valves were found perforation and insuffientia in all patients and pannus in17 patients.
    结果:术中所有病人均证实存在瓣膜穿孔及关闭不全,17例发现瓣缘有赘生物。
  15. With MRI, bone erosions were observed in 20 patients, polyarticular synovial hyperplasia, joint effusion, and periarticular inflammation tissue was seen in all 30 patients, pannus was showed in 23 patients. Abnormalities of tendons and ligaments were also diagnosed in 7 patients on MRI.
    MRI显示20例关节面骨侵蚀,所有30例均可见关节滑膜增厚、关节积液以及关节周围炎性渗出,23例可见血管翳形成,7例合并关节周围肌腱韧带损伤。
  16. The pathogenesis of RA is still unclear. However, a large number of studies have demonstrated that activation of many kinds of immunocytes, formation of pannus, participation of cytokine and mediators of inflammation play important roles in RA.
    RA的病因及发病机制至今尚未完全阐明,但是大量研究证实:多种免疫活性细胞的活化,血管翳的形成,细胞因子、炎症介质的共同参与是造成疾病发生发展的重要原因。
  17. It is generally accepted that synovitis is the basic pathogenetic lesion of RA in which there are extensive synovial cell proliferation, pannus formation and presence of a series of increased amounts of cytokines and inflammation mediators like prostaglandin E2 ( PGE2).
    已知RA的基本病变系滑膜炎,其中有广泛的滑膜细胞增殖、血管翳形成及大量的细胞因子和炎症介质如前列腺素E2(prostaglandinE2,PGE2)。
  18. Under the cover of the pannus cartilage surface can be seen clearly the organization of the cartilage surface degeneration and necrosis.
    在血管翳覆盖下的软骨表层可见明显的软骨表层组织的变性、坏死。
  19. The Fundamental pathological change is the joint synovial chronic inflammation leading to formation of pannus, which destroys cartilage, bone and surrounding tissues.
    它的基本病理改变为关节滑膜慢性炎症形成侵袭性血管翳,破坏软骨、骨与周围组织。
  20. The manifestations are peripheral joint persistent synovial inflammation, synovial organization hyperplasia, inflammatory cells invasion, pannus formation, thereby cause the cartilage and bone destruction, cause joint deformities and loss of function, eventually lead to different degree of disability.
    表现为外周关节持续性滑膜炎、滑膜组织增生、炎性细胞侵润、血管翳形成,继而引起软骨及骨的进行性破坏,造成关节畸形和功能丧失,最终导致不同程度的残废。
  21. Corneal neovascularization may play a part in the corneal wound healing, but a chronic corneal neovascularization can result in vascular pannus, and damage of corneal microenvironment and corneal immune privilege.
    虽然角膜新生血管对角膜损伤修复起到一定作用,但是长期慢性的角膜血管新生会形成角膜翳,破坏角膜正常微环境,使眼前节免疫赦免减弱或消失,影响视力健康。
  22. The pathological characteristic of RA includes synovial hyperplasia and inflammation, pannus formation, in the end bone erosion, cartilage loss and joint destruction. If left untreated, it would lead to joint damage and disability.
    主要病理特征是关节滑膜的慢性炎症、增生,形成血管翳,造成关节软骨、骨和关节囊的破坏,如果不及时治疗,最终导致关节畸形和功能丧失。
  23. After two months of implantation, a pannus developed slowly from the textile suturing ring on both sides with a fragile layer of endothelial like-cells on the fibrosa side that extended slowly.
    瓣膜植入2个月后,血管翳慢慢地从缝合环的两面长出。在纤维层表面,宿主自身内皮细胞开始生长。
  24. Sacroiliitis probably initiated with bone marrow inflammation, followed by pannus formation, subchondral bony plate destruction, and cartilage degeneration/ erosion, eventually leading to fibrosis, ossification and joint fusion.
    SIJ炎可能自骨髓开始,继而血管翳形成,软骨下骨板破坏,软骨变性、破坏,最后纤维化、骨化而导致关节融合。