periportal
英
美
网络 门脉三角区周围; 门脉周围; 门部扩展到汇管区; 门静脉周围的; 门静脉周的
双语例句
- Endogenous ketogenesis was1.3-times higher in the perivenous zone than in periportal hepatocytes.
肝静脉周围肝细胞的内源性酮体生成比门静脉周围肝细胞高1.3倍。 - Zonation of ketone body and glucose metabolism was studied in hepatocyte preparations enriched with cells from the periportal or the perivenous zone of the liver lobule.
用大鼠肝脏门静脉或肝静脉周围的肝细胞来研究葡萄糖和酮体生成的区域分布。 - Out of the 18 cases, TTV DNAs was detected in 8 cases ( 44.4%), in which the positive cells were scattered in the intralobular areas and more aggregated in the periportal and necrotic areas.
原位杂交阳性8例（444%）。TTV阳性表达细胞散在分布于肝小叶内，坏死区及汇管区旁较密集。 - Duplex Doppler Sonography for Caroli's Diseases "Periportal Fibrosis Type"
Caroli'S病门静脉周围纤维化型的双功能超声诊断 - Stimulation of gluconeogenesis by administration of gluconeogenic substrates resulted in 1.7-2.1-times higher rates of glucose synthesis in the periportal cells than in perivenous hepatocytes.
给予刺激葡萄糖异生的底物，门静脉周围肝细胞的葡萄糖合成则增加1.7－2.1倍。 - Conclusions The oval cells originate from bile duct of the periportal regions.
结论卵圆细胞起源于汇管区的胆管上皮； - Results: All the MR images of 13 patients showed widening of periportal space with diffuse abnormal long T_1 long T_2 signal;
结果：13例患者均在脂肪抑制T2WI上出现门静脉周围间隙增宽，并伴有长T1、长T2信号； - To detect the telomerase and vascular endothelial growth factor ( VEGF) activity in liver of mice with advance stage of schistosomiasis japonica and to discuss the relationship between periportal cirrhosis and hepatocarcinoma.
目的检测晚期血吸虫病肝组织的端粒酶和血管内皮细胞生长因子（VEGF）的活性，探讨日本血吸虫病肝硬化与恶性变的相关性。 - This hepatic progenitor cells located in periportal areas, fiber spacing, periportal zone adjacent hepatic parenchyma, and inflammatory pseudo-lobular borders.
这种肝前体细胞定位于汇管区、纤维间隔、汇管区旁肝实质、假小叶及炎症边界。 - Were seen scattered in the lamivudine therapy mouse liver periportal lymphocyte infiltration, fibrous tissue hyperplasia, with mild fatty degeneration of liver cells around the central vein.
而拉米夫定治疗小鼠肝组织汇管内见散在的淋巴细胞浸润，无纤维组织增生，但中央静脉周围肝细胞轻度脂肪变性。 - Obvious liver structure of rats in model group, hepatic lobule structure was not clear, liver cells arranged in disorder, liver cell degeneration, necrosis, hepatic periportal inflammatory cell infiltration, liver cell diffuse vacuolar degeneration.
模型组大鼠肝脏结构破坏较明显，肝小叶结构欠清，肝细胞索排列紊乱，肝细胞变性、溶解坏死、肝汇管区炎性细胞浸润明显，肝细胞呈弥漫性空泡样变。 - Modeled after the saline group: diffuse large areas of necrosis, cell lysis, the lobular structure obscure, lobular and periportal inflammatory cell infiltration mainly by lymphocytes and macrophages, residual edema and degeneration of liver cells, There were no significant changes with the model group.
建模后生理盐水组：出现弥漫性的大片坏死，细胞溶解，小叶结构模糊不清，小叶内及汇管区有淋巴细胞和巨噬细胞为主的炎细胞浸润，残存肝细胞水肿变性，与模型组无明显变化。