phosphatidylinositol
英
美
网络 磷脂酰肌醇; 肌醇磷脂; 磷酸肌醇; 磷酯酰肌醇; 磷脂酸肌胺
双语例句
- The effect of adenosine and its analogues on the phosphorylation of phosphatidylinositol in pig erythrocyte ghosts was investigated.
本文研究了腺苷及其类似物对猪红细胞膜上磷脂酰肌醇磷酸化的影响。 - The influence on VEGF expression by phosphatidylinositol 3-kinase signaling pathway in hepatocellular carcinoma cells
肝癌细胞中PI3K信号通路对VEGF表达的影响 - Effects of alcohol and high-fat diet on the expression of phosphatidylinositol 3-kinese in rat adipose tissue
酒精联合高脂饮食对大鼠脂肪组织磷脂酰肌醇3激酶表达的影响 - Radiosensitization of phosphatidylinositol 3-kinase inhibitor on esophageal cancer cell line Eca-109 and its mechanism
PI3K抑制剂对食管癌Eca-109细胞株放射增敏作用及机制 - Effect of phosphatidylinositol 3-kinase on inducible nitric oxide synthase in bronchiole epithelial cells of asthmatic rat
磷脂酰肌醇3激酶对哮喘大鼠气道上皮细胞诱导型一氧化氮合酶表达的调控作用 - Interleukin-1 Receptor Associated Kinase 1 and Phosphatidylinositol 3 Kinase Regulate the NF-κ B Activation Induced by IL-1
IL-1R相关激酶-1和磷脂酰肌醇3-激酶协同调控IL-1诱导的NF-κB活化 - Objective To investigate the roles of phosphatidylinositol 3-kinase ( PI3K) and p42/ p44 MAPK in the regulation of transcription of vascular endothelial growth factor ( VEGF) in hepatocellular carcinoma ( HCC) cells.
目的研究磷脂酰肌醇3激酶（PI3K）和p42/p44丝裂源激活的蛋白激酶（MAPK）在肝癌细胞血管内皮生长因子（VEGF）转录调控中的作用。 - There is obvious difference in phosphatidylinositol 4-kinase protein level between the experimental and control groups.
免疫组织化学结果显示：与正常组相比，缺血组磷酸肌醇4激酶蛋白表达水平明显升高。 - So this article focused on the change of endothelial function and the disorder of insulin/ phosphatidylinositol 3-kinase ( PI3-K) pathway to explore the mechanisms related to blood pressure elevation in the insulin-resistant state, at the level of animal model and cell culture.
因此，本文将从动物试验、细胞培养两个方面通过对内皮功能及胰岛素磷脂酰肌醇3-激酶（PI3-K）信号转导途径的研究来探讨胰岛素抵抗状态下血压升高的机制。 - The separation of phosphatidylinositol ( PI), phosphatidylethanolamine ( PE), phosphatidylcholine ( PC) from soybean phospholipids has been researched by using low-pressure column chromatography under isocratic elution.
通过实验探讨了使用低压制备色谱柱在等度洗脱方式下分离大豆磷脂中的磷脂酰肌醇（PI）、磷脂酰乙醇胺（PE）、磷脂酰胆碱（PC）等成分。 - Active focal adhesion kinase regulates cell adhesion, migration, proliferation and differentiation by interacting with Src family kinase, phosphatidylinositol 3 kinase, cytoskeletal proteins, Graf and adoptor proteins through the phosphorylated tyrosines and the proline rich sequences.
通过磷酸化酪氨酸位点和富脯氨酸序列，活化的焦点粘着激酶与细胞骨架蛋白、Src族激酶、磷酸肌醇3激酶、Graf以及多种衔接子蛋白相互作用，调节细胞的粘附、迁移、增殖和分化。 - [ Objective] To investigate the expression and activity of phosphatidylinositol 3-kinase in skeletal muscle of insulin resistance mice.
目的了解老年胰岛素抵抗小鼠胰岛素刺激的骨骼肌细胞磷脂酰肌醇-3激酶（PI3-K）表达和活性变化。 - The post receptor signal transduction consists of a series of receptor associated tyrosine protein kinase activated intracellular protein phosphorylation and dephosphorylation reactions, such as the phosphorylation of insulin receptor substrate 1 ( IRS 1), Shc, Ras and phosphatidylinositol 3 kinase ( PI3 K).
受体后的信号转导经过了一系列磷酸化和去磷酸化等途径，其中有胰岛素受体底物1（IRS1）、Shc蛋白、Ras蛋白以及磷酸肌醇3激酶（PI3K）等的参与。 - In addition, it was discovered that phosphatidylinositol 3-kinase/ Akt pathway is involved in the HIF-1 trans-activation.
另外，PI-3K/AKT信号通路也参与HIF-1转录活性的调节。 - Role of the phosphatidylinositol 3-kinase/ AKT signaling pathway in culturing neural stem cells in vitro
体外神经干细胞培养过程中蛋白激酶磷脂酰肌醇-3激酶/AKT信号途径的作用 - Objective To investigate the changes of phosphatidylinositol 4-kinase activity in myocardium and red blood cells during prolonged myocardial ischemia.
目的观察大鼠心肌缺血后心肌细胞和红细胞膜上磷酸肌醇4激酶活性的变化，探讨其在心肌缺血过程中的调控机理。 - BACKGROUND& OBJECTIVE: Adenosine plays an essential role in cell s signal transduction, including inhibiting the activity of phosphatidylinositol 4-kinase, up-regulating the activity of adenylate cyclase and the concentration of cAMP, and inhibiting platelet actin polymerization.
背景与目的：腺苷作用于细胞信号转导系统，可抑制磷脂酰肌醇4-激酶活性、升高腺苷酸环化酶活性和细胞内cAMP水平、抑制血小板肌动蛋白聚合等。 - C-CBL Is the bridge molecule linking BCR/ ABL to phosphatidylinositol 3 kinase
c-CBL是连接BCR/ABL与磷脂酰肌醇-3激酶的桥梁 - It is known that through binding to the tyrosine kinase receptor RET, the glial cell line-derived neurotrophic factor ( GDNF) activates two intracellular signaling pathways: the phosphatidylinositol 3 kinase ( PI3K) pathway and the Ras/ Raf/ MEK/ Erk pathway.
胶质细胞系源性神经营养因子（GDNF）可以通过跨膜的酪氨酸蛋白激酶受体RET激活细胞内两条信号通路，即磷脂酰肌醇3激酶（PI3K）信号通路和Ras/Raf/MEK/Erk信号通路。 - Expression of phosphatidylinositol 3-kinase in skeletal muscle of insulin resistance mice
老年胰岛素抵抗小鼠骨骼肌细胞磷脂酰肌醇-3激酶的表达 - Conclusion The inhibitor of phosphatidylinositol 3 ′-kinase path could up-regulate the apoptosis of gastric cancer cells.
结论磷脂酰激酶3通路抑制剂渥曼青霉素可以增强胃癌细胞凋亡的发生。 - Conclusion That IP inhibits inflammatory cascades through phosphatidylinositol 3-kinase signal transduction pathway may be one of its protective mechanism.
结论通过磷酸肌醇3激酶信号传导通路抑制炎性反应可能是缺血预处理的保护机制。 - Blocking phosphatidylinositol metabolism partially inhibited these changes.
阻断磷脂酰肌醇代谢后可部分抑制上述生化参数的变化。 - Regulation of phosphatidylinositol 3-kinase in skeletal muscle and the effect of exercise on it
磷酯酰肌醇3激酶在骨骼肌中的调节以及运动对它的影响 - Objective To explore the relationship between thyroid adenoma and phosphatidylinositol 3-kinase/ protein kinase B ( PI3K/ Akt) signaling pathway.
目的探讨甲状腺腺瘤疾病与磷脂酰肌醇3激酶/蛋白激酶B（PI3K/Akt）信号转导途径之间的关系。 - Objective: To study the activation of Akt and its upstream molecule, phosphatidylinositol 3 kinase ( PI3K) induced by E-cadherin-mediated cell-cell adhesion and their effect on ovarian cancer cell proliferation.
目的研究E-钙粘素介导的细胞粘附对卵巢癌细胞Akt及其上游的磷脂酰肌醇3-激酶（phosphatidylinositol3-Kinase，PI3K）信号的激活及对卵巢癌细胞增殖的作用。 - The phosphatidylinositol turnover of S180 cell membrane was inhibited strongly when PPS acted on cell membranes at suitable condition.
将PPS与S180细胞膜在适当条件下一同温育，发现PPS干扰膜的肌醇磷脂代谢，明显抑制磷脂酰肌醇转换。 - The Activation of Phosphatidylinositol 3-kinase and Extracellular Signal-regulated Kinase in Human Breast Cancer and the Analysis of Correlation
磷脂酰肌醇3-激酶与胞外信号调节激酶在人乳腺癌中的活化及其相关性分析 - The Pten tumor suppressor gene encodes a phosphatase that negatively regulates the phosphatidylinositol 3-kinase ( PI3K) pathway activity.
抑癌基因Pten编码磷酸酶，负调控磷脂酸肌醇3-激酶（PI3K）途径的活性。