retinoid
英
美
网络 类视黄醇; 视黄醛衍生物; 类视色素; 维A酸; 维甲酸
COCA.33591
双语例句
- The Effects of Receptors of WGA, RCA-I and ECL on the Development of Cleft Palate Induced by Excess Retinoid Acid in KM Mice
WGA、RCA-I和ECL凝集素受体在过量维甲酸致昆明小鼠腭裂发生中的作用 - Given the severity of acne and evidence of inflammatory lesions, the optimal regimen for LM is a combination of a topical retinoid and an antimicrobial agent.
考虑到痤疮的严重程度以及炎性皮损的存在，LM的最佳办法是局部联合用维甲酸类和抗微生物制剂。 - Retinoid X receptor Agonists Inhibit High-Glucose-Induced Oxidative Stress in Human Endothelial Cells
视黄醇类X受体激动剂抑制高糖诱导内皮细胞氧化应激 - Purpose: To analyse the differentiation features of embryonic stem cells which were induced by the supernatant of retinal cells and retinoid acid ( RA).
目的：探讨经过初步诱导的拟胚体在视黄酸和视网膜细胞培养上清液作用下的分化特征。 - I see a huge difference in patients who've used a retinoid for years& they look younger than their counterparts, and their skin is smoother and more resilient.
我看到了使用了几年维甲酸治疗的病人的巨大差别，他们比同龄人看看年轻，他们的皮肤更光滑，更有弹性。 - Objective To study the alteration of retinoid X receptor α ( RXR α) with the treatment of tazarotene in active psoriasis vulgaris and investigate the mechanism.
目的研究他扎罗汀治疗进行期寻常型银屑病前后异维甲酸受体α（RXRα）的变化，并探讨其作用机制。 - Methods The N-CoR expression in 16 kinds of leukemia cell lines and the changes of acute promyelocytic leukemia cell lines with retinoid acid were detected using RT-PCR.
方法：RT-PCR半定量检测16种急性非淋巴细胞白血病细胞株中N-CoR的基因表达及急性早幼粒细胞白血病细胞株在维A酸作用过程中的变化。 - HB-EGF may play a key role in the early pathogenesis of psoriasis and the psoriatic keratinocyte growth. Retinoid acid may regulate the expression of HB-EGF in psoriatic epidermis by some mechanism, which inhibits proliferation of epidermis and induces normal differentiation.
HB-EGF可能在银屑病发病的早期及维持银屑病表皮角质形成细胞的增殖中起关键作用，维A酸可能通过某种机制来调节表皮HB-EGF的表达，从而抑制银屑病表皮增殖并诱导正常分化。 - Phosphorylated JNK mediated apoptosis induced by All Trans Retinoid Acid in human retinoblastoma cell line
磷酸化JNK介导全反式维甲酸诱导视网膜母细胞瘤细胞凋亡 - AIM: To investigate the feasibility and effect of directly differentiation of embryonic stem cells ( ESC) into neural cells induced by retinoid acid ( RA) without embryonic body ( EB) culture period in vitro.
目的：探讨不经拟胚体（EB）培养阶段，应用维甲酸（RA）在体外直接将胚胎干细胞（ESC）诱导分化为神经细胞的可行性和效果。 - AtRA exerts its effects by binding the retinoic acid receptor ( RAR) and retinoid X receptor ( RXR).
己证实atRA通过核内维甲酸受体（RAR）和维甲类X受体（RXR）的激活而起作用。 - All Trans Retinoid Acid Inhibit Cell Growth in Human Retinoblastoma Cells Via Phosphorylation of JNK
全反式维甲酸通过磷酸化JNK抑制视网膜母细胞瘤细胞生长 - 【 Methods 】 The levels of vitamin A and retinoid binding proteins were detected by the trace fluorescence assay and ELISA respectively.
【方法】采用微量荧光法和ELISA方法分别测定人群血浆中维生素A的含量和视黄醇结合蛋白的浓度。 - Expression of retinoid acid receptors in embryonal adrenal gland and its relationships to neuroblastoma
胚胎肾上腺分化过程视黄酸受体表达及神经母细胞瘤发生的关系探讨 - Methods: SK-N-SH cell morphological changes induced by LTD4, pranlukast and LTD4+ pranlukast were observed with retinoid acid ( RA) as the positive control.
方法：以维A酸为阳性对照，观察LTD4、普鲁司特、LTD4+普鲁司特诱导SK-N-SH细胞形态学变化； - Study on the binding of retinoid X receptor with β 2 rare
维甲类X受体与维甲酸反应元件β2RARE结合的研究 - Function of apoptosis induced by derivatives of retinoid acid on tumor cells
维甲酸衍生物对肿瘤细胞的凋亡诱导作用 - Objective: To investigate the effect of all trans retinoid acid ( ATRA) on bone cell proliferation and differentiation.
目的：探讨全反式视黄酸（ATRA）对体外培养的新生大鼠成骨细胞增殖与分化的影响。 - Objective To investigate the regulatory effects of peroxisome-proliferator activated receptor γ ( PPAR γ) and retinoid X receptor ( RXR) agonists on the aromatase activity in human ovarian granulosa cells.
目的探讨过氧化酶体增殖物激活受体γ（PPARγ）和维甲类X受体（RXR）激动剂对人卵巢颗粒细胞芳香化酶活性的调节作用。 - Effects of a Novel Retinoid on Cyclin D1 and E in Breast Cancer Cells
新型维甲酸对乳腺癌细胞周期蛋白D1、E表达的影响 - Effects of all-trans retinoid acid on the carcinoma MEC-1 cells
全反式维甲酸对人粘液表皮样癌MEC-1细胞表型的影响 - Method: Determine retinoid in self-emulsifying preparation by HPLC and evaluate dissolution behavior compared with market capsule.
方法：采用HPLC法测定维甲酸RA自乳化制剂的药物含量，与市售胶囊比较，考察RA自乳化制剂体外溶出行为。 - Another, retinoid resistance has been identified in the treatment of a variety of malignant cells.
另外维甲酸抵抗在恶性肿瘤的治疗中也被证实。 - The most stable RA is the third-generation retinoid – ATRA.
其中以第三代维甲酸ATRA结构最为稳定。 - The enhancement of ligand-dependent degradation of retinoid receptors by SIP suppressed retinoids-induced gene expression which caused the induction of retinoid resistance in tumor cells.
S1P通过增强维甲酸受体的配体依赖性降解作用，进而抑制维甲酸类药物促进靶基因的转录，致肿瘤细胞对维甲酸类药物产生耐药性。 - Loss of retinoid receptors expression happens frequently in the development of carcinogenesis and induction of resistance to retinoids.
维甲酸受体的表达缺失是癌症发生过程中的常发与重要事件，这可能导致肿瘤对维甲酸类药物产生耐药性。