Metasilicate acid can improve telangiectasia, decrease incidence of cardiovascular and avoid skin diseases, gynecopathy& diabetes. 偏硅酸:能改善血管扩张,降低心血管发病率,可防治皮肤病、妇科病、糖尿病。
Color Doppler ultrasound in diagnosis of hereditary hemorrhagic telangiectasia with hepatic involvement 彩超诊断累及肝脏的遗传性出血性毛细血管扩张症
Treatment of facial telangiectasia by using XL-radiofrequency skin ablation apparatus XL-射频皮肤治疗仪消融在毛细血管扩张治疗中的应用
Because people sleep after muscle relaxation, telangiectasia, porokeratosis large, central temperature regulation function, easy to catch a cold or other diseases; 因为人睡后肌肉松弛、毛细血管扩张、汗孔张大,体温调节中枢功能减退、易患感冒或其他疾病;
Ultra-high field 3.0T MR susceptibility weighted imaging in diagnosis of intracerebral capillary telangiectasia 超高场3.0T磁共振磁敏感加权成像诊断颅内毛细血管扩张症
Objective To investigate the value of different MR sequences in diagnosing intracerebral capillary telangiectasia ( ICT). 目的探讨磁共振成像不同序列对颅内毛细血管扩张症的影像诊断价值。
Objective To localize the pathogenic gene of hereditary hemorrhagic telangiectasia ( HHT) in a Chinese family of Han Nationality. 目的定位一个中国汉族遗传性出血性毛细血管扩张症家系的致病基因。
The establishment of facial telangiectasia animal modle by ultraviolet irradition 紫外线诱导的面部毛细血管扩张动物模型的建立
Objective To investigate the characteristics of the cytogenetic anomalies of ataxia telangiectasia. 目的探讨共济失调毛细血管扩张症的细胞遗传学异常特点。
PVA microspheres embolization: a case of hepatic involvement in hereditary hemorrhagic telangiectasia PVA微球栓塞肝脏遗传性出血性毛细血管扩张症1例
Hereditary hemorrhagic telangiectasia resulted from a nonsense mutation Arg 479 Stop in the ALK-1 gene ALK-1基因无义突变Arg479Stop导致的遗传性出血性毛细血管扩张症
ATM gene mutations in Chinese patients with ataxia-telangiectasia 中国人共济失调毛细血管扩张症ATM基因突变研究
The genetic disorder ataxia telangiectasia ( AT) is characterized by immunodeficiency, progressive cerebellar ataxia, gonadal abnormalities, radiosensitivity, and cancer predisposition. 基因缺陷所致共济失调-毛细血管扩张症(AT)表现为免疫缺陷、进行性共济失调、性腺发育异常、辐射敏感和易患癌症等。
By electron microscope, different degree of glial cell swelling and telangiectasia on the experimental side was seen. But, on the control side, no obvious glial cell swelling or telangiectasia was seen. 在电镜下,实验侧脑组织见不同程度的胶质细胞肿胀及毛细血管扩张,而对照侧未见明显的胶质细胞肿胀或毛细血管扩张。
Symptomatic children with hereditary hemorrhagic telangiectasia: A pediatric center experience 有症状的遗传性出血性毛细血管扩张症患儿的临床和遗传学特征:来自儿科中心的经验
Ultrasonographic Application in Diagnosing Hereditary Hemorrhagic Telangiectasia Involving Liver 超声在遗传性出血性毛细血管扩张症肝受累中的应用价值
Conclusion Endoscopic heat probe is an effective method in colon telangiectasia. 结论内镜下热极治疗结肠毛细血管扩张症是一种微创有效安全的方法。
Analysis of angiogenesis related proteins and its implication in type-2 hereditary hemorrhagic telangiectasia Ⅱ型遗传性出血性毛细血管扩张症血管生长发育相关蛋白质分析及意义
Caspase 3 mediated Cleavage of Ataxia Telangiectasia Mutated Protein in Induction of Apoptosis 毛细血管扩张性共济失调突变蛋白在细胞凋亡过程中被Caspase┐3降解
The Treatment of Nosebleed Caused by Hereditary Hemorrhagic Telangiectasia 中西医结合治疗遗传性出血性毛细血管扩张症致鼻出血疗效观察
Methods Seventeen patients with colon telangiectasia were treated by endoscopic heat probe. 方法对17例结肠毛细血管扩张症患者应用内镜下热极治疗。
Ataxia Telangiectasia ( AT) as a genetic disorder results from single gene mutation, characterized by radiosensitivity, progressive nerve degeneration, immunodeficiency, premature aging and cancer predisposition. 共济失调性毛细血管扩张综合症(AT)是一种单基因突变引起的遗传性疾病,表现为高辐射敏感性、进行性神经退变、免疫缺陷、早衰及易患癌症等。
Objective: To study the treatment of nosebleed caused by hereditary hemorrhagic telangiectasia ( HHT). 目的:探讨遗传性出血性毛细血管扩张症所引起鼻出血的治疗方法。
Mutation analysis of the ATM gene in two Chinese patients with ataxia telangiectasia 2例华裔共济失调毛细血管扩张症患者的ATM基因突变分析
Objective To explore the effecting of endoscopic heat probe in colon telangiectasia. 目的探讨内镜下热极治疗毛细血管扩张症的疗效。
It has now been confirmed that Ataxia Telangiectasia mutated ( ATM) protein plays a crucial role in the regulation of cell cycle which is regulated by corresponding cyclin. 细胞周期由相应的周期蛋白进行调控,现已证实共济失调毛细血管扩张突变蛋白(AtaxiaTelangiectasiamutatedprotein,ATMprotein)在这一过程中发挥着关键的调控作用。
RCM indicated that a week after treatment, telangiectasia and edema were observed. After the inflammation has subsided, collagens were mesh-shape aligned. RCM检测示治疗后在1周可见毛细血管扩张、水肿等炎性反应,1个月后可见到胶原纤维呈网状排列。
Part of the ear appeared focal interstitial small abscess or acute inflammation, inflammation with telangiectasia, vascular endothelial cell structural integrity, no swelling, degeneration, necrosis and hyperplasia. 部分耳廓间质出现灶性小脓肿或急性炎症改变,炎症部位有毛细血管扩张,血管内皮细胞结构完整,无肿胀、变性、坏死及增生。